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Since LGD 4033 is a suppressive compound, testosterone suppression while on cycle is a natural and obvious side effectof testosterone treatment. Since we already know that all of the other testosterone treatments are not the problem, why should we worry about DHT? There is no natural DHT to take into account, so what is causing some people to experience problems taking DHT (and more specifically testosterone)? The following are 2 theories on what causes problems in DHT treatment. The first is a theoretical explanation, the second is a more practical theory which is used by men on cycle to reduce DHT. The theoretical explanation In this example DHT has an effect on DHT-less testosterone, so in order to be in a reduced state of DHT we need a new DHT – we can't just keep adding in DHT, we need a new one. There are two factors that work on DHT, both of which are related to its "hormone". The First Factor – DHT-less testosterone The first factor is called DHT-less testosterone, and it causes DHT, but doesn't directly bind to testosterone. You can think of this as being a generic form of testosterone substitute. It is not made by testosterone itself, because it does not have effects on the body which cause higher testosterone levels to occur, so it is only created directly by the body. While this would help treat men who are in testosterone deficient conditions like hypogonadism, it is not a practical means to treat people currently on cycle and/or testosterone blockers. Furthermore, men who suffer from DHT deficiency are likely to suffer from hyperandrogenism (abnormal levels of circulating testosterone), which is only a temporary symptom; a more permanent problem is likely to come. The second factor – Testosterone blockers Testosterone blockers, anabolic agents, were introduced in the 1970s, as a way to lower DHT levels through direct exposure to the substance in order to make them inactive. This worked very well, and testosterone was back on the market within a 20 year period. Unfortunately it appears that it has been doing more harm than good. The first problem is with testosterone itself. When testosterone is exposed to air it reacts with oxygen to form water, called DHT. When it is exposed to heat it reacts with the oxygen in the air, to form water, called DHT-L, then after being broken down by the liver it forms a new molecule by using an enzyme called 5α-reductase, but after many years testosterone blockers cause DHT to Similar articles:

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